Welcome

Welcome

A unifying description of the root cause of cancer ensues.   You are reading it here as a result of encouragement by cancer patients, friends and family.  Flemming Rasmussen – Mar 2014

Pause for thought –

Sound science chases the statistical outliers; a great example are spontaneous/radical remissions of cancer. Knowing that such a reversal is possible brings euphoric emotions, failing to identify any consistent mechanism behind them perplexing, and knowing they arise extremely infrequently is sobering. Reflecting on this phenomenon results in a surreal realization the body can heal itself WITHOUT conventional medical interventions despite significant cancer burden. But how?

Each patient carries his own doctor inside him. They come to us not knowing that truth.
We are at our best when we give the doctor who resides in each patient a chance to go to work.” 
Dr. Albert Schweitzer

Our thesis overview 

Things about cancer you rarely (or perhaps never) consider, let alone hear about

Things about cancer you rarely (or perhaps never) consider, let alone hear about

One common denominator present in every single cancer (neonatal to geriatric, and all types) is the intimate relationship between our inherited genes with our personally unique cellular microenvironments. Before advancing the cancer specific link, it is imperative to appreciate the role of lifestyle and diet on our genes.  In the slide above, the picture of the fish1 illustrates that a resulting phenotype is profoundly influenced by both the genes (expected) and the cytosol (oft ignored); earlier surrogate mother studies vividly show the same influences.  This analogy’s foundation boils down to different gene expressions, differences entirely due to the surrounding and nurturing environment. Shifting this concept to a human example, focus on the transformation over a lifetime from conception to adulthood. Had you ever considered that your genome remained essentially unchanged throughout this journey, faithfully repeating itself in each successive cell division (the march from 1 to ~40 trillion cells!) and that your gene expressions are integrally linked to the microenvironment of each cell’s cytosol? The cytosol’s gradual transformation over your lifetime (from conception’s maternal influences through to an adult’s insidious dietary surpluses and deficiencies, coupled with exposures through other means) can be viewed as a dynamic matrix that causes ALL of our genes’ expression at any height of wellness or depth of disease.

Slide1

This is the dance; it is a dance that can’t occur without both the genes and the cytosol ‘communicating’ in some manner and doing the best they can to survive; cancer is an increasingly predictable cellular consequence to a dynamic environmental insult!

Essentially all of the mainstream cancer research has seemingly been unaware or brushed over this critical aspect of a cell’s health and has rather opted to focus on singular contributing but subordinate roles and downstream effects of observed changes in pH, degraded mitochondria functioning, or our genome and its unwanted genetic expression.

Closing Thanks

The thrust of this website centers on tending to a compromised cellular environment. Accordingly, our expectation is that within this website you will find our understanding of cancer not only interesting, but enlightening, hopeful and meaningful.  Thank you!

1. Sun, Y. H., S. P. Chen, et al. (2005). “Cytoplasmic Impact on Cross-Genus Cloned Fish Derived from Transgenic Common Carp (Cyprinus carpio) Nuclei and Goldfish (Carassius auratus) Enucleated Eggs