Welcome (updated 10 Sep 2017)

A seldom articulated view of cancer ensues.   You are reading it here as a result of encouragement by cancer patients, friends and family.  Flemming Rasmussen – Mar 2014

“It ain’t what you don’t know that gets you into trouble. It’s what you know for sure that just ain’t so.”  author unknown but oft attributed to Mark Twain

Expanding on the theme of this quote, this website digs into what we see as the three main impediments in the mainstream cancer research (each of which could be described as a different view of the same issue).  They are as follows:

a.    Combined, DNA and the surrounding environment rule the roost – Reflect on how often you read about the former with nary a mention of the latter

b.   Failure to assimilate reasons behind pH shifts; these shifts are ubiquitous in cancer – Appreciating the role of pH in cancer is hindered further by an omission of assimilating the Dr Peter Stewart three independent pH variables with the mainstream pH theories, and

c.   The element composition of a cell/the body matters

Where the trail has led us

In all of cancer’s complexity, missing is an understanding of what occurs when a ‘typical’ body assimilates upwards of 60 elements yet it only needs 17-24 of so-called “essential elements” to live.  The adjacent ‘elementome’ cartoon provides a view of  the origin of each person’s cellular soup and suggests a causal link to the buzzwords like genes, proteins, and fats that dominate most conversations about health.   The impact of having surpluses or deficiencies of elements in our bodies is proving to be an extremely fruitful area of study on our cancer quest.

The cancer narrative often glosses over the sources of elements /molecules in our bodies, the very foundation for each and every molecule that drives the biochemical reactions in our body.

A factor in every single cancer (neonatal to geriatric, and all types) is the intimate relationship between our inherited genes with our personally unique cellular microenvironment. The role of lifestyle and diet on our genes is profound; for example,  in the poster below, the picture of the fish1 illustrates that the resulting ‘offspring’ is influenced by both the genes (expected) and the cytosol (oft ignored).  ‘Surrogate mother’ studies dating back to the 1950s show the same influences.  This analogy’s foundation boils down to altering gene expressions, modifications entirely due to each cells surrounding local environment.  Part of each cell division, the cytosol’s composition gradually transforms over our lifetime (from conception’s maternal influences through to an adult’s dietary and lifestyle influences – some of which are insidious).


Take away is that from conception to now, our body is exclusively the product of diet/lifestyle


The cytosol of our cells is a dynamic matrix that is implicated in our genes’ expression at any height of wellness or depth of disease.  This is the dance; it is a dance that can’t occur without both the genes and the cytosol ‘communicating’; cancer is an increasingly predictable cellular consequence to a dynamic diet and lifestyle insult!


Mainstream cancer research largely brushes over this critical aspect of a cell’s ‘health’ and has rather opted to focus on singular contributing but subordinate roles and downstream effects of observed changes in pH, degraded mitochondria functioning, or our genome and its unwanted genetic expression.

Closing & Thanks

With the appreciation our personal diet and lifestyle contributions are major factors in all cancers, the thrust of this website also casts a lens on first determining the make-up, and then as applicable, tending to a compromised cellular environment. A work in progress,  this website presents a view of cancer with an aim to suggest new,  safe and broadly effective treatment options should arise from such an understanding.  Complementing this effort, we also advance a pH theory envisioned to confront cancer burdens rapidly and is eluded to in the side-page comments; more on this soon.

Thank you!

1. Sun, Y. H., S. P. Chen, et al. (2005). “Cytoplasmic Impact on Cross-Genus Cloned Fish Derived from Transgenic Common Carp (Cyprinus carpio) Nuclei and Goldfish (Carassius auratus) Enucleated Eggs